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Thursday, September 5, 2013

Dysentery - Nursing Diagnosis and Interventions

1. Nursing Diagnosis for Dysentery : Imbalanced Nutrition: less than body requirements
related to: inadequate intake and output

Goal: nutritional needs are met

Expected outcomes:
  • Increased appetite.
  • Increased or normal weight according to age.

Nursing Interventions for Dysentery:
1. Discuss and explain about the diet restrictions of patients (high fiber foods, fatty and water is too hot or cold)
R / high fiber, fat, water is too hot / cold can stimulate irritate the stomach and intestinal tract.

2. Create a clean environment, away from the smell of the odor or litter, serve food in a warm state.
R / situation comfortable, relaxed to stimulate appetite.

3. Provide hours of rest (sleep) and reduce excessive activity.
R / Reducing energy consumption is excessive

4. Monitor intake and output within 24 hours.
R / Knowing the amount of output can merencenakan amount of food.

5. Collaboration with other health care team:
a. Nutritional therapy: A diet high in calories and high in protein, low in fiber, milk.
b. medications or vitamins (A)
R / Containing substances necessary for the growth process.


2. Nursing Diagnosis for Dysentery: Imbalanced Body Temperature
related to: the impact of infection secondary to diarrhea.

Goal: no increase in body temperature

Expected outcomes:
  • Body temperature within normal limits (36-37,5 C)
  • There are no signs of infection (rubur, dolor, calor, tumor, fungtio leasa)

Nursing Interventions for Dysentery:
1. Monitor body temperature every 2 hours.
R / Early detection of abnormal changes in body function (an infection)

2. Give warm compresses.
R / stimulate heat regulating center to reduce the production of body heat

3. Collaboration of antipyretic
R / Stimulate the heat regulating center in the brain.


3. Nursing Diagnosis for Dysentery: Risk for Impaired Skin Integrity: perianal
related to: increased frequency of bowel movements (diarrhea)

Goal: skin integrity is not compromised

Expected outcomes :
  • No irritation: redness, blisters, hygiene maintained.
  • Families are able to demonstrate perianal care properly.

Nursing Interventions for Dysentery:
1. Discuss and explain the importance of keeping the beds.
R / Hygiene prevent the proliferation of germs.

2. Demontrasikan and involve families in the treatment of perianal (if wet clothing and replace the bottom as well as the base).
R / Preventing skin iritassi unexpected because kelebaban and stool acidity.

3. Adjust the position of sleep or sit with an interval of 2-3 hours.
R / Smooth vascularization, reducing the emphasis that long so did not happen ischemia and irritation.


4. Nursing Diagnosis for Dysentery: Anxiety: children
related to: invasive measures

Goal: the client is able to adapt

Expected outcomes:
  • Want to receive care measures, the client seems quiet and no fuss

Nursing Interventions for Dysentery:
1. Involve the family in performing maintenance actions.
R / initial approach to the child through the mother or family.

2. Avoid the wrong perception on nurses and hospitals.
R / reduce the fear of the child to the nurse and the hospital environment.

3. Give kudos if the client would be given care and treatment measures.
R / increase the child's confidence will courage and ability.

4. Make contact as often as possible and do communication both verbal and non-verbal (touching, fondling, etc.).
R / Love and the introduction of self saying nurses would menunbuhkan sense of security on the client.

5. Give children toys as sensory stimuli.

Nursing Care Plan for Dysentery

Dysentery is a inflammatory disorder of the intestine, especially the colon, which results in severe diarrhea containing mucus and / or blood in the stool. If left untreated, dysentery can be fatal.

Cause of Dysentery

Dysentery is usually caused by a bacterial or protozoan infection or infestation of parasitic worms, but can also be caused by chemical irritants or viral infection. The two most common causes are infection with a bacillus of the Shigella group, and infestation by an amoeba, Entamoeba histolytica. When it is caused by a bacillus called bacillary dysentery, and when it is caused by an amoeba called amoebic dysentery.

Symptoms of Dysentery

Dysentery symptoms can last for five days or even more. For some cases, the symptoms may be mild, while others suffer from severe diarrhea and vomiting or potentially cause dehydration. The following symptoms when exposed to dysentery:
  • Flatulence
  • Pain in the abdomen
  • Bloody diarrhea
  • Nausea, with or without vomiting

However, if the infection is severe, people may experience other symptoms caused by dehydration:
  • Decreased urine production
  • Dry skin and mucous membranes
  • Excessive thirst
  • Fever and chills
  • Muscle spasms
  • Limp
  • Weight loss
  • Yellowish white mucus

In cases of chronic dysentery, no effects after an acute attack. In severe cases, the body temperature will rise to 40 degrees Celsius to 40.6 degrees Celsius.


Prevention of Dysentery

Dysentery is spread as a result of poor hygiene. To minimize the risk of these conditions, then it should be done on the prevention of dysentery:
  • Avoid swallowing water in swimming pools or recreational water sources
  • Make sure you drink water that has been purified or boiled water
  • Drink bottled water when traveling
  • Wash your hands with anti-bacterial soap after using the bathroom, changing diapers, before preparing and eating food.
  • Avoid sharing towels with others
  • Wash clothing or eating utensils of an infected person.


Nursing Care Plan for Dysentery

Assessment

1. Identity

Noteworthy is the age. Episodes of diarrhea occurred in the first 2 years of life. Highest incidence is the age group 6-11 months. Most bacteria stimulate gut immunity against infection, it helps explain the decline insidence disease in older children. At the age of 2 years or more of active immunity begins to form. Most cases are due to an intestinal infection and asymptomatic enteric bacteria spread mainly clients are not aware of the infection. Economic status also influential, especially from the diet and treatment.

2. Main complaint
Defecation is more than 3 x

3. History of present illness
Defecation greenish yellow color, mixed with mucus and blood or mucus alone. Watery consistency, frequency is more than 3 times, spending time : 3-5 days (acute diarrhea), more than 7 days (prolonged diarrhea), more than 14 days (chronic diarrhea).

4. Past medical history
Never had diarrhea before, to those on long-term antibiotics or corticosteroids (candida albicans changes from saprophyte to parasite), food allergies, respiratory infections, UTI, OMA measles .

5. History of Nutrition
At toddler age children are given food as in adults, the portion given 3 times per day with additional fruit and milk. Malnutrition in toddler age children are particularly vulnerable. Way better food management, food hygiene and sanitation, hand-washing habits.

6. Family health history
There is one family that has diarrhea.

7. Environmental Health History
Food storage at room temperature, lacking hygiene, neighborhood .

8. Growth and development history
a. growth
  • Weight gain since age 1 -3 years ranged between 1.5-2.5 kg (average of 2 kg), a body length of 6-10 cm (mean 8 cm) per year.
  • The increase in head circumference: 12cm 2 cm in the first year and second year and so on.
  • Teething 8 pieces: additional milk teeth; first molars and canines, totaling 14-16 pieces
  • Eruption of teeth: molars perama menusul canines.
b. development
  • Psychosexual stages of development according to Sigmund Freud.

5 Nursing Diagnosis related to Acute Diarrhea

Acute diarrhea is defined as the discharge of bowel movements once or more in the form of a liquid in a day and lasted less than fourteen days. Diarrhea is a condition that is not normal stool expenditure, usually characterized by increased volume, dilution and frequency of bowel movements more than 3 times a day with or without mucus and blood.

The presence of food that can not be absorbed by the intestinal lumen will cause osmotic pressure in the cavity resulting in increased intestinal absorption of water and electrolytes into the intestinal cavity. Excessive intestinal cavity contents will stimulate the intestine to release it, causing diarrhea. Non-pathogenic bacteria in the intestinal lumen (often called intestinal flora) can cause diarrhea. Normally through the process of fermentation of non-pathogenic intestinal bacteria metabolize a variety of substrates, especially of food substances with the end result of fatty acids and gases.

The anaerobic metabolism will provide additional energy for the body. Due to intestinal stasis, obstruction and malnutrition lead to an increase in the number of non-pathogenic bacteria so that the fermentation process food substances produce metabolites that are not wanted by the body. For example: lactose (from milk) is a good food for the non-pathogenic bacteria. Lactose will be fermented to produce gas and cause gastric distension.

Result of the high concentration of lactose causes an osmotic pressure in the intestinal lumen increases. Hyperosmolar state will absorb water from the intra-cellular, followed by an increase in intestinal peristalsis resulting in diarrhea.

5 Nursing Diagnosis related to Acute Diarrhea

1. Nursing Diagnosis : Deficient Fluid Volume
related to excessive fluid loss through the stool or vomit
characterized by :

Subjective data :
  • thirst , nausea , anorexia .

Objective data :
  • Inadequacy of oral fluid intake
  • Negative balance between intake and output
  • Weight loss
  • Dry mucous membranes
  • Decreased urine output
  • Decrease in skin turgor
  • Increase in serum sodium

2. Nursing Diagnosis :  Imbalanced Nutrition: less than body requirements
related to loss of fluids through diarrhea, inadequate intake is
characterized by :

Subjective data :
  • Family clients reported a portion of food that is spent.
  • Abdominal cramps.

Objective data :
  • Weight loss below ideal body weight.
  • Upper arm circumference below the ideal.
  • Anemic conjunctiva.
  • Anorexia.
  • Muscle weakness.
  • Decrease in serum albumin.

3. Nursing Diagnosis : Risk for infection
related to microorganisms that penetrate the gastrointestinal tract .

4. Nursing Diagnosis : Impaired skin integrity : perianal
related to irritation from diarrhea
characterized by :

Subjective data :
  • Changes in comfort : pain, itching

Data obtektif :
  • Damage to the skin layer ( dermis ) : lesions and skin irritation due to diaper.
  • Perianal area moist and redness.

5. Nursing Diagnosis : Anxiety / fear
relatde to separation from parents , unfamiliar environment , stressful procedure
characterized by :

Subjective data :
  • Reported feelings of anxiety , fear

Objective data :
  • Restless
  • Focus on yourself
  • Less eye contact
  • Choleric
  • Tremor
  • Facial tension
  • Increased respiratory and pulse
  • Sweat

Monday, September 2, 2013

Activity Intolerance NIC NOC

Insufficient physiological or psychological energy to endure or complete required or desired daily activities

Defining Characteristics:
  • Verbal report of fatigue or weakness,
  • abnormal heart rate or blood pressure response to activity,
  • exertional discomfort or dyspnea,
  • electrocardiographic changes reflecting dysrhythmias or ischemia

Related Factors:
  • Bed rest or immobility;
  • generalized weakness;
  • sedentary lifestyle;
  • imbalance between oxygen supply and demand

NOC

Suggested NOC Labels
  • Endurance
  • Energy Conservation
  • Activity Tolerance
  • Self-Care: Activities of Daily Living (ADLs)
Client Outcomes
  • Participates in prescribed physical activity with appropriate increases in heart rate, blood pressure, and breathing rate; maintains monitor patterns (rhythm and ST segment) within normal limits
  • States symptoms of adverse effects of exercise and reports onset of symptoms immediately
  • Maintains normal skin color and skin is warm and dry with activity
  • Verbalizes an understanding of the need to gradually increase activity based on testing, tolerance, and symptoms
  • Expresses an understanding of the need to balance rest and activity
  • Demonstrates increased activity tolerance

NIC

Suggested NIC Labels
  • Energy Management
  • Activity Therapy

Nursing Interventions and Rationales

1. Determine cause of activity intolerance (see Related Factors) and determine whether cause is physical, psychological, or motivational.
Determining the cause of a disease can help direct appropriate interventions.

2. Assess client daily for appropriateness of activity and bed rest orders.
Inappropriate prolonged bed rest orders may contribute to activity intolerance. A review of 39 studies on bed rest resulting from 15 disorders demonstrated that bed rest for treatment of medical conditions is associated with worse outcomes than early mobilization (Allen, Glasziou, Del Mar, 1999).

3. Minimize cardiovascular deconditioning by positioning clients as close to the upright position as possible several times daily.
The hazards of bed rest in the elderly are multiple, serious, quick to develop, and slow to reverse. Deconditioning of the cardiovascular system occurs within days and involves fluid shifts, fluid loss, decreased cardiac output, decreased peak oxygen uptake, and increased resting heart rate (Resnick, 1998).

4. If appropriate, gradually increase activity, allowing client to assist with positioning, transferring, and self-care as possible. Progress from sitting in bed to dangling, to chair sitting, to standing, to ambulation.
Increasing activity helps to maintain muscle strength, tone, and endurance. Allowing the client to participate decreases the perception of the client as incapable and frail (Eliopoulous, 1998).

5. Ensure that clients change position slowly. Consider using a chair-bed (stretcher-chair) for clients who cannot get out of bed. Monitor for symptoms of activity intolerance.
Bed rest in the supine position results in loss of plasma volume, which contributes to postural hypotension and syncope (Creditor, 1993).


6. When getting clients up, observe for symptoms of intolerance such as nausea, pallor, dizziness, visual dimming, and impaired consciousness, as well as changes in vital signs.
Heart rate and blood pressure responses to orthostasis vary widely. Vital sign changes by themselves should not define orthostatic intolerance (Winslow, Lane, Woods, 1995).


7. Perform range-of-motion exercises if client is unable to tolerate activity.
Inactivity rapidly contributes to muscle shortening and changes in periarticular and cartilaginous joint structure. These factors contribute to contracture and limitation of motion (Creditor, 1994).

8. Refer client to physical therapy to help increase activity levels and strength.

9. Monitor and record client's ability to tolerate activity: note pulse rate, blood pressure, monitor pattern, dyspnea, use of accessory muscles, and skin color before and after activity. If the following signs and symptoms of cardiac decompensation develop, activity should be stopped immediately (ACSM, 1995):
  • Excessive fatigue
  • Lightheadedness, confusion, ataxia, pallor, cyanosis, dyspnea, nausea, or any peripheral circulatory insufficiency
  • Onset of angina with exercise
  • Palpitations
  • Dysrhythmia (symptomatic supraventricular tachycardia, ventricular tachycardia, exercise-induced left bundle block, second- or third-degree atrioventricular block, frequent premature ventricular contractions)
  • Exercise hypotension (drop in systolic blood pressure of more than 10 mm Hg from baseline blood pressure despite an increase in workload, when accompanied by other evidence of ischemia)
  • Excessive rise in blood pressure (systolic greater than 220 mm Hg or diastolic greater than 110 mm Hg); NOTE: these are upper limits; activity may be stopped before reaching these values
  • Inappropriate bradycardia (drop in heart rate greater than 10 beats/min) with no change or increase in workload
  • Increased heart rate above the prescribed limit

10. Instruct client to stop activity immediately and report to physician if experiencing the following symptoms: new or worsened intensity or increased frequency of discomfort, tightness, or pressure in chest, back, neck, jaw, shoulders, and/or arms; palpitations; dizziness; weakness; unusual and extreme fatigue; excessive air hunger.
These are common symptoms of angina and are caused by a temporary insufficiency of coronary blood supply. Symptoms typically last for minutes as opposed to momentary twinges. If symptoms last longer than 5 to 10 minutes, the client should be evaluated by a physician (McGoon, 1993). The client should be evaluated before resuming activity (Thompson, 1988).

11. Allow for periods of rest before and after planned exertion periods such as meals, baths, treatments, and physical activity. Rest periods decrease oxygen consumption (Prizant-Weston, Castiglia, 1992).

12. Observe and document skin integrity several times a day. Activity intolerance may lead to pressure ulcers.
Mechanical pressure, moisture, friction, and shearing forces all predispose to their development (Resnick, 1998).

13. Assess urinary incontinence related to functional ability. Assess independent ability to get to the toilet and remove and adjust clothing.
The loss of functional ability that accompanies disease often leads to continence problems. The cause may not be the person's bladder instability but his or her ability to get to the toilet quickly (Nazarko, 1997).

14. Assess for constipation.
Impaired mobility is associated with increased risk of bowel dysfunction, including constipation. Constipation increases the risk of urinary tract infection and urge incontinence (Nazarko, 1997).

15. Consider dietitian referral to assess nutritional needs related to activity intolerance.
Severe malnutrition can lead to activity intolerance. Dietitians can recommend dietary changes that can improve the client's health status (Peckenpaugh, Poleman, 1999).

16. Refer the cardiac client to cardiac rehabilitation for assistance in developing safe exercise guidelines based on testing and medications.
Cardiac rehabilitation exercise training improves objective measures of exercise tolerance in both men and women, including elderly patients with coronary heart disease and heart failure. This functional improvement occurs without significant cardiovascular complications or other adverse outcomes (Wenger et al, 1995).

17. Ensure that the chronic pulmonary client has oxygen saturation testing with exercise. Use supplemental oxygen to keep oxygen saturation 90% or above or as prescribed with activity.
Supplemental oxygen increases circulatory oxygen levels and improves activity tolerance (Petty, Finigan, 1968; Casaburi, Petty, 1993).

18. Monitor a chronic obstructive pulmonary disease (COPD) client's response to activity by observing for symptoms of respiratory intolerance such as increased dyspnea, loss of ability to control breathing rhythmically, use of accessory muscles, and skin tone changes such as pallor and cyanosis.

19. Instruct and assist COPD clients in using conscious controlled breathing techniques such as pursing their lips and diaphragmatic breathing.
Training clients with COPD to slow their respiratory rate with a prolonged exhalation (with or without pursed lips) helps control dyspnea and results in improved ventilation, increased tidal volume, decreased respiratory rate, and a reduced alveolar-arterial oxygen difference. This breathing pattern not only helps relieve dyspnea but can improve the ability to exercise and carry out ADLs (Mueller, Petty, Filley, 1970; Casaburi, Petty, 1993).

20. Provide emotional support and encouragement to client to gradually increase activity.
Fear of breathlessness, pain, or falling may decrease willingness to increase activity.

21. Refer the COPD client to a pulmonary rehabilitation program.
Pulmonary rehabilitation has been shown to improve exercise capacity, walking ability, and sense of well-being (Fishman, 1994).

22. Observe for pain before activity. If possible, treat pain before activity, and ensure that client is not heavily sedated.
Pain restricts the client from achieving a maximal activity level and is often exacerbated by movement.

23. Obtain any necessary assistive devices or equipment needed before ambulating client (e.g., walkers, canes, crutches, portable oxygen).
Assistive devices can increase mobility by helping the client overcome limitations.

24. Use a walking belt when ambulating a client who is unsteady.
With a walking belt the client can walk independently, but the nurse can provide support if the client's knees buckle.

25. Work with client to set mutual goals that increase activity levels.


Geriatric

1. Slow the pace of care. Allow client extra time to carry out activities.

2. Encourage families to help/allow elder to be independent in whatever activities possible. Sometimes families believe they are assisting by allowing clients to be sedentary.
Encouraging activity not only enhances good functioning of the body's systems but also promotes a sense of worth by providing an opportunity for productivity (Eliopoulous, 1997).

3. When mobilizing the elderly client, watch for orthostatic hypotension accompanied by dizziness and fainting.
Orthostatic hypotension is common in the elderly as a result of cardiovascular changes, chronic diseases, and medication effects (Mobily, Kelley, 1991).


Home Care Interventions

1. Begin discharge planning as soon as possible with case manager or social worker to assess need for home support systems and the need for community or home health services.

2. Assess the home environment for factors that precipitate decreased activity tolerance: presence of allergens such as dust, smoke, and those associated with pets; temperature; energy-intensive activity patterns; and furniture placement. Refer to occupational therapy if needed to assist the client in restructuring the home and activity of daily living patterns.
Clients and families often estimate energy requirements inaccurately during hospitalization because of the availability of support.

3. Teach the client/family the importance of and methods for setting priorities for activities, especially those having a high energy demand (e.g., home/family events).

4. Provide client/family with resources such as senior centers, exercise classes, educational and recreational programs, and volunteer opportunities that can aid in promoting socialization and appropriate activity.
Social isolation can contribute to activity intolerance.

5. Discuss the importance of sexual activity as part of daily living. Instruct the client in adaptive techniques to conserve energy during sexual interactions.
Families may make unsafe choices for sexual activity or place added stress on themselves trying to cope with this issue without proper support or teaching.

6. Instruct the client and family in the importance of maintaining proper nutrition and rest for energy conservation and rehabilitation.

7. Refer to medical social services as necessary to assist the family in adjusting to major changes in patterns of living.

8. Assess the need for long-term supports for optimal activity tolerance of priority activities (e.g., assistive devices, oxygen, medication, catheters, massage), especially for hospice patients. Evaluate intermittently.
Assessments ensure the safety and appropriate use of these supports.

9. Refer to home health aide services to support the client and family through changing levels of activity tolerance. Introduce aide support early. Instruct the aide to promote independence in activity as tolerated.
Providing unnecessary assistance with transfers and bathing activities may promote dependence and a loss of mobility (Mobily, Kelley, 1991).

10. Be aware of increased risk of bone fracture even after muscle strength is normalized, especially in osteopenic-prone individuals such as estrogen-deficient women and the elderly.
Reduction in weight bearing muscle activity during bed rest invariably produces significant changes in calcium balance and, in weeks, changes in bone mass (Bloomfield, 1997)

11. Allow terminally ill clients and their families to guide care.
Control by the client or family promotes effective coping.

12. Provide increased attention to comfort and dignity of the terminally ill client in care planning. For example, oxygen may be more valuable as a support to the client's psychological comfort than as a booster of oxygen saturation.



Client/Family Teaching

1. Instruct client on rationale and techniques for avoiding activity intolerance.
2. Teach client to use controlled breathing techniques with activity.
3. Teach client the importance and method of coughing, clearing secretions.
4. Instruct client in the use of relaxation techniques during activity.
5. Help client with energy conservation and work simplification techniques in ADLs.
6. Teach client the importance of proper nutrition.
7. Describe to client the symptoms of activity intolerance, including which symptoms to report to the physician.
8. Explain to client how to use assistive devices or medications before or during activity.
9. Help client set up an activity log to record exercise and exercise tolerance.

Tuesday, July 30, 2013

Ineffective Tissue Perfusion related to Encephalitis

Nursing Diagnosis: Ineffective Tissue Perfusion related to increased intracranial pressure.

Goals:
  • Patient's neurological status returned to the state before the illness.
  • Increased patient awareness and sensory function.

Outcomes:
  • Vital signs within normal limits.
  • Reduced headache pain.
  • Increased awareness.
  • No signs or loss of increased intracranial pressure.

Intervention:

1. Total bedrest patients, with supine sleeping position without a pillow.
Rationale: Changes in inta-cranial pressure will be able to mislead the risk for brain herniation.

2. Monitor signs of neurological status with GCS.
Rational: It can reduce further brain damage.

3. Monitor vital signs such as blood pressure, pulse, temperature, respiration and caution in systolic hypertension.
Rational: In normal circumstances autoregulation maintains a state of altered systemic blood pressure fluctuation. Autoregulation failure will cause a cerebral vascular damage can be manifested by an increase followed by a decrease in systolic and diastolic pressure. While the increase in temperature can describe the course of infection.

4. Monitor intake and output
Rational: Hyperthermia can lead to increased IWL and increase the risk of dehydration, especially in patients who are not aware, and nausea were lower intake by mouth

5. Help the patient to limit vomiting, coughing. Instruct the patient to exhale when moving or turning in bed.
Rationale: Activity vomiting or coughing can increase intracranial and intra-abdominal pressure. Exhale when moving or changing position can protect themselves from the effects of Valsalva.

6. Give fluids per infusion with strict attention.
Rationale: Minimize the burden of vascular and fluctuations in intracranial pressure, fluid and fluid vetriksi can reduce cerebral edema.

7. Monitor blood gas analysis of oxygen delivery when needed.
Rational: The possibility of acidosis is accompanied by the release of oxygen at the cellular level may lead to the occurrence of cerebral ischemic.

8. Provide appropriate therapy such as physician advice: Steroids, Aminofel, Antibiotics
Rational Therapy given to decrease capillary permeability.
Lowering of cerebral edema
Lowered metabolic cells / consumption and seizures.

Monday, July 29, 2013

Nursing Care Plan for Meniere's Disease

Definition of Meniere's Disease

Meniere's Disease is a chronic disorder of the semicircular canal and the labyrinth of the inner ear, appear to be associated with over-production of endolymph in the inner ear (Elizabeth Corwin J: 2009).

Ménière's disease is a disease that affects the inner ear endolymphatic fluid pressure in the deeper parts of the ear that is responsible for balance and hearing function. Symptoms usually affect these functions and may differ from person to person. (Ananya Mandal: 2013)

Type of Meniere's Disease

1. Vestibular Meniere's disease
Vestibular Meniere's disease is characterized by episodic vertigo with respect to the pressure in the ears without cochlear symptoms.
Signs and symptoms:
  • Merely episodic vertigo.
  • Decrease in vestibular response or no response total pain in the ear.
  • There was no cochlear symptoms.
  • There was no hearing loss objective.
  • Later may develop symptoms and signs of cochlear.

2. Classic Meniere's disease,
Signs and symptoms:
  • Complained of vertigo
  • Fluctuating sensorineural hearing loss
  • Tinnitus
  • Cochlear Meniere's disease

3. Cochlear Meniere's disease
Cochlear Meniere's disease identified with progressive sensorineural hearing loss with respect to tnitus and pressure in the ear without any findings or vestibular symptoms.
Signs and symptoms:
  • Fluctuating hearing loss
  • Aural pressure or full feeling
  • Tinnitus
  • Hearing loss seen in test results
  • There was no vertigo
  • Normal vestibular labyrinth test
  • Later will suffer symptoms and signs of vestibular (Nn: 2011)


The degree of severity of Meniere's disease;

1. Grade I:
Early symptoms include vertigo accompanied by nausea and vomiting. Vagal disorders such as pale and sweating may occur. Before the attack of vertigo symptoms, the patient may feel a sensation in the ear, which lasted for 20 minutes to several hours. Among, the patient is normal.

2. Grade II:
Hearing loss deepened and fluctuate. Symptoms of the low-frequency sensorineural hearing loss.

3. Grade III:
Hearing loss is no longer fluctuating but progressive worsening. This time on both ears so deaf patients as having total. Vertigo began to decrease or disappear. (Nuzulul Zulkarnain Haq: 2009)


Etiology of Meniere's disease

The exact cause of Meniere's disease, until now not known with certainty, many experts have different opinions. Until now considered the cause of disease is caused by a disturbance in the physiology of the system, known as endolymph endolymph hydrops, a condition where the amount of endolymph fluid that resulted in an abrupt increase of the scale dilatation media. However, the cause of hydrops endolymph has yet to be ascertained.
There is some contention as to the cause of hydrops, among others:
  • Increasing hydrostatic pressure at the arterial end.
  • Reduced osmotic pressure in the capillaries.
  • Increasing the osmotic pressure of the extra-capillary space.
  • Way out sac endolimfatikus clogged, resulting in accumulation endolimfa.
  • Middle ear infection.
  • Upper respiratory tract infection.
  • Head trauma.
  • Consumption of foods that contain caffeine and high salt.
  • Consumption of aspirin, alcohol, and cigarettes were prolonged.
  • Herpesviridae virus infection group.
  • Hereditary.


The following will explain the cause of Meniere's disease is considered to trigger:

1. Herpes virus (HSV)
Herpes viruses are found in patients with Meniere's. Once there was a report that 12 of the 16 patients there Meniere herpes simplex virus DNA in endolimfatikusnya sac. In addition it has been reported also in Meniere's patients who were given antiviral therapy are improved. But this assumption has not been proven entirely because they still need further research.

2. Hereditary
In the study found 1 in 3 patients had a parent who suffered from Meniere's disease as well. Hereditary predisposition is considered to have a relationship with anatomical abnormality or abnormalities in the channel endolimfatikus immune system.

3. Allergy
In patients with Meniere found that 30% of them have food allergies. Relationship between allergies with Meniere's disease are as follows: endolimfatikus sac may be the target organ of mediators that are released in the body when holding a reaction to certain foods. Antigen-antibody complexes may interfere with the ability of the sac endolimfatikus filtration. There is a relationship between allergy and viral infection that causes hydrops of endolimfatikus sac.

4. Head trauma
Scarring caused by trauma to the inner ear can be considered disturbing the hydrodynamic flow of endolimfatikus. This assumption is reinforced by the Meniere's patients who have a history of temporal bone fracture.

5. Autoimmune
There is also a presumption of experts stating that endolymph hydrops is not a cause of Meniere's disease. It is said by Honrubia in 1999 and Rauch in 2001 that the autopsy study found endolymph hydrops in 6% of people who do not suffer from Meniere's disease. Much research is now focused on immunologic function in endolimfatikus sac. Some experts argue Meniere's disease caused by an autoimmune disorder. Brenner conducted a study in 2004 said that in about 25% of patients with Meniere's disease is also found to thyroid autoimmune diseases. Additionally in 2002 Ruckenstein also getting in approximately 40% of patients with Meniere's disease obtained positive results in the examination of the blood such as autoimmune arthritis factor, antiphospholipid antibodies and Anti Sjoegren. (Nuzulul Zulkarnain Haq: 2009)

Wednesday, July 24, 2013

Nursing Concepts and Care Plan for Mental Retardation (MR)

Mental retardation (MR) is a condition in which a person has the mental capacity is insufficient. Mental retardation is a subnormal intellectual function abnormalities occur during development and is associated with one or more disorders of maturation, learning and social adjustment.

Mental retardation is defined as weakness / inability cognitive appeared in childhood (before the age of 18 years) is characterized by the function under normal intelligence (IQ 70-75 or less), and accompanied by at least two other limitations in the following areas: speech and language; self-care skills, ADL; social skills; using community facilities, health and safety; functional academic, work and relax, etc..

Clinical manifestations

Clinical manifestations of mental retardation, among others:
1. Cognitive impairment (pattern, thought process).
2. The slow reception skills and language expression.
3. Failed to get past the main stages of development.
4. Head circumference is above or below normal (sometimes larger or smaller than normal size).
5. Possibility of slow growth.
6. Possibility of abnormal muscle tone (more frequent weak muscle tone).
7. Possibility of dysmorphic features.
8. Delays in fine and gross motor development.

Pathophysiology

Mental retardation refers to the real limitations of daily living function. Mental retardation include weakness or cognitive disability that appears in childhood (before age 18 years) were characterized by below-normal intelligence function (IQ 70 to 75 or less) and with other limitations in adaptive functioning at least two areas: speaking and language, abilities / skills of self-care, homemaking, social skills, use of community facilities, self-direction, health and safety, functional academic, leisure and work. Cause of mental retardation can be classified into prenatal, perinatal and post-natal. Diagnosis of mental retardation established early in childhood.

Complication

1. Cerebral palsy
2. Seizure disorders
3. Psychiatric disorders
4. Impaired concentration / hyperactivity
5. communication deficits
6. constipation

Prevention

1. Increase healthy brain development and the provision of care and an environment that stimulates growth.
2. Should focus on the biological health and early life experiences of children living in poverty in terms of prenatal care, regular health monitoring and family support services.


Nursing Care Plan for Mental Retardation (MR)

A. Assessment

The assessment consists of a comprehensive evaluation of the shortcomings and strengths associated with the adaptive skills; communication, self-care, social interaction, use of facilities in the community self-direction, health care and safety, functional academic, recreational skill formation, and tranquility.

B. Nursing Diagnosis

1. Impaired growth and development related to cognitive dysfunction.
2. Impaired verbal communication related to cognitive dysfunction.
3. Risk for injury related to aggressive behavior imbalance of physical mobility.
4. Impaired Social Interaction related to difficulty speaking / social adaptation difficulties.
5. Interrupted family processes related to having a child with mental retardation.
6. Self care deficit related to changes in physical mobility / lack of developmental maturity.

C. Intervention

1. Assess the factors causing impaired child development.
2. Identification and use of educational resources to facilitate optimal child development.
3. Provide consistent care.
4. Increase communication verbal and tactile stimulation.
5. Give simple instructions and repeat.
6. Give positive reinforcement on child outcomes.
7. Encourage children to do their own maintenance.
8. Difficult child behavior management.
9. Encourage children to socialize with the group.
10. Create a safe environment.

D. Education on Parents

1. Each stage of child development for ages.
2. Support parental involvement in child care.
3. Anticipatory guidance and management face a difficult child behavior.
4. Inform existing educational facilities and groups.

E. Expected results
1. Children to function optimally the relevant level.
2. Families and children are able to use coping with challenges due to disability.
3. Families are able to obtain the resources community facilities.
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